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The manifestations of symptoms in premenstrual syndrome (PMS) can vary. Typically, three main symptom patterns are observed:
1. Type 1: In this most common type, symptoms begin in the late premenstrual period, i.e., in the days leading up to menstruation. This is the classic presentation initially described for the syndrome.
2. Type 2: Symptoms start with the ovulation period, spontaneously subside or disappear within one or two days, and then reappear in the premenstrual period.
3. Type 3: Symptoms begin with ovulation and continuously increase throughout the luteal phase, reaching their peak. This varying course and pattern typically recur similarly in most menstrual cycles for women experiencing this condition.
Causes of Premenstrual Syndrome (PMS)
While the exact causes of PMS are not fully understood, it is believed to be associated with increased sensitivity in the central nervous system, hormonal imbalances, and changes in brain chemicals. In women who are overly sensitive to the natural cyclical changes occurring due to hormonal functioning, these fluctuations can trigger biochemical events related to PMS in the central nervous system and other target tissues.
Serotonin hormone plays a critical role in the processes leading to the emergence of PMS symptoms in the central nervous system. Fluctuations in serotonin, a brain chemical crucial for mood regulation, can exacerbate PMS symptoms. Studies conducted on women diagnosed with PMS have revealed several differences in their serotonergic systems compared to women without PMS.
Progesterone is primarily produced in the ovaries. While progesterone itself has the potential to induce anxiety, some of its metabolites (products) are known to have anxiolytic effects. Progesterone can also increase serotonin reuptake, leading to changes in the serotonin cycle.
What are the Types of Premenstrual Syndrome (PMS)?
1. Type 1: In this most common type, symptoms begin in the late premenstrual period, i.e., in the days leading up to menstruation. This is the classic presentation initially described for the syndrome.
2. Type 2: Symptoms start with the ovulation period, spontaneously subside or disappear within one or two days, and then reappear in the premenstrual period.
3. Type 3: Symptoms begin with ovulation and continuously increase throughout the luteal phase, reaching their peak. This varying course and pattern typically recur similarly in most menstrual cycles for women experiencing this condition.
Causes of Premenstrual Syndrome (PMS)
While the exact causes of PMS are not fully understood, it is believed to be associated with increased sensitivity in the central nervous system, hormonal imbalances, and changes in brain chemicals. In women who are overly sensitive to the natural cyclical changes occurring due to hormonal functioning, these fluctuations can trigger biochemical events related to PMS in the central nervous system and other target tissues.
Serotonin hormone plays a critical role in the processes leading to the emergence of PMS symptoms in the central nervous system. Fluctuations in serotonin, a brain chemical crucial for mood regulation, can exacerbate PMS symptoms. Studies conducted on women diagnosed with PMS have revealed several differences in their serotonergic systems compared to women without PMS.
Progesterone is primarily produced in the ovaries. While progesterone itself has the potential to induce anxiety, some of its metabolites (products) are known to have anxiolytic effects. Progesterone can also increase serotonin reuptake, leading to changes in the serotonin cycle.